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tmj Dislocation

 Temporomandibular Joint Dislocation

Hypermobility disorders of the temporomandibular joint  

(TMJ) present in two major forms: 

1. Dislocation 

2. Subluxation [1] 

The differences between dislocation and subluxation have  

been highlighted in Box 64.1. 

The condyle may be displaced either anteriorly, posteriorly, medially, or laterally of which anterior dislocation is the  

most common [1]. The incidence of TMJ dislocation constitutes about 3% of the dislocations occurring in other joints of  

the body with female predilection. The reported incidence of  

TMJ dislocation is 7% with a preponderance in people in the  

second and third decades [2]

1. Acute 

2. Chronic 

Dislocation recurring more than once is termed as chronic  

recurrent dislocation. The term chronic protracted dislocation is used to describe dislocation persisting for more than 1  

month, while dislocation present for more than 6 months is  

called extra-long-standing dislocation [4]. 

Based on the direction of displacement [2],

Classifcation (Box 64.2) 

Dislocation has been classifed by numerous methods. Rowe  

and Killey [3] based it on the duration of the dislocation episode as:

• Anterior 

• Posterior 

• Lateral 

• Superior 

Anterior dislocation is the most common type of dislocation due the weakness of the capsule in the anterior region.

Posterior dislocation occurs following fracture of the  

external auditory canal and skull base. Allen and Young  

classified lateral dislocation into Type I and Type II. This  

type of dislocation occurs in mandibular trauma  

(Fig. 64.1a, b): 

• Type I refers to lateral subluxation. 

• Type II indicated lateral and superior dislocation into the  

temporal fossa [2]. 

Superior dislocation results when condyle is pushed into  

the middle cranial fossa accompanied by glenoid fossa fracture. Small and round-shaped condyle is predisposed to this  

type of dislocation [2]. 

Etiopathogenesis (Table 64.1) 

A multitude of causes have been described in the etiopathogenesis of TMJ dislocation including congenital,  

iatrogenic, anatomical aberrations, spontaneous, pharmacological, neurological, neuromuscular, etc. Proper diagnosis of the etiology is important to institute problemspecifc treatment

Daily activities which involve wide mouth opening such as  

laughing, yawning, and biting may induce TMJ dislocation. It  

may also occur spontaneously during epileptic seizures, vomiting, yawning, and singing. Trauma is another cause which  

might cause posterior, superior, and lateral dislocation in addition to anterior dislocation [5]. Iatrogenic causes include dental procedures which require wide mouth opening for  

prolonged time, intubation procedures, gastrointestinal endoscopy, and laryngoscopy/bronchoscopy. Anatomical aberrations such as small condyle, underdeveloped glenoid fossa,  

shallow/steep articular eminence and laxity of ligaments and  

capsule are more prone for dislocation. 

Predisposing risk factors include connective tissue disorders such as Ehlers-Danlos disease and Marfan’s syndrome  

which predispose to laxity of the joint and hypermobility.  

Muscle spasms occur in neurodegenerative or neurodysfunctional diseases, namely, Huntington disease, Parkinson disease, multiple sclerosis, muscle dystrophies, or dystonias. 

Medications which induce dislocation are antipsychiatric  

(phenothiazines) and antiemetic (metoclopramide) drugs which  

produce unwanted extrapyramidal reactions which eventually  

lead to muscular imbalance attributed to dislocation. 

Reduced vertical dimension due to loss of posterior teeth  

in advanced age may also predispose an individual to dislocation [5]. 

Though various theories of pathogenesis have been  

described in literature, the most accepted was muscular incoordination during mandibular movements. In the initialstages of mouth closure, elevators are activated prior to the  

relaxation of depressors mainly lateral pterygoid which pulls  

the condyle forward. This initial dislocation facilitates the  

further dislocation [5].

 

Clinical Features  

(Mentioned in Box  

64.3; Figs. 64.2a, b, 64.3, 64.4a, b,  

and 64.5a, b)


Investigations 

• Orthopantomogram (OPG) (open and closed) (Fig. 64.6) 

This is the commonly used screening modality for the  

examination of TMJ.  Morphology of condyle, articular  

eminence, and joint space can be evaluated. Open mouth  

OPG shows the position of the condyle in relation to the  

articular eminence. 

• TMJ tomogram 

Open and closed mouth TMJ images can be obtained in  

different slices. 

• Computed tomography(CT) 

Evaluation of the morphology of osseous TMJ components—condyle, articular eminence and the glenoid  

fossa—are better assessed with CT. 

• Cone beam computed tomography CBCT 

CBCT facilitates accurate measurement of condylar  

height, width, and length as well as inclination of articular  

eminence. 

• Magnetic resonance imaging (MRI) 

MRI demonstrates the soft tissue morphology, particularly disc shape, displacement, and effusion of the joint  

frequently associated with dislocation. 

• Electromyography (EMG) 

EMG evaluates the activity of the muscles which may be  

hypoactive, normoactive, or hyperactive. 

• Ultrasonography (USG) 

Thickness and length of the muscles can be evaluated  

both at rest and clenching by USG. 


cle spasm persisting for longer duration. In diffcult situations, reduction can be facilitated with the help of local  

anesthesia, conscious sedation, and general anesthesia.  

Following reduction, a Barton’s bandage, chin strap, or intermaxillary fxation is advised for 3–6 weeks to prevent further  

dislocation. Several reduction techniques have been  

employed with varying rates of success. 

Management of Dislocation 

Acute Dislocation 

Reducing the dislocated condyle poses a great challenge.  

Reduction is more complicated with the accompanied muscle spasm persisting for longer duration. In diffcult situations, reduction can be facilitated with the help of local  

anesthesia, conscious sedation, and general anesthesia.  

Following reduction, a Barton’s bandage, chin strap, or intermaxillary fxation is advised for 3–6 weeks to prevent further  

dislocation. Several reduction techniques have been  

employed with varying rates of success.

Hippocratic/Nelaton’s Technique  

(Fig. 64.7) 

This is the conventional method of reduction of acute dislocation in which physician stands in front of the patient, with  

the thumb placed either on the external oblique ridge or on  

the lower molars and other fngers positioned along the lower  

border of the mandible. A steady downward, backward, and  

superior force should be given to reduce the dislocated  

condyle. The thumb should be protected either with a plastic  

splint or gauze wrapped around it to prevent injury to the  

thumb while reducing dislocation. 

64.6.1.2 Gag Refex [3] 

Gag refex is induced by probing the soft palate using mouth  

mirror. In alert individuals, this refex relaxes the lateral pterygoid muscle through coordinated neuromuscular activities  

which reduces dislocation in natural way. 

64.6.1.3 Wrist Pivot Method [7] (Fig. 64.8) 

This method utilizes existing myospasm of the elevators for  

reduction. The thumb is placed under the chin, while other  

fngers are placed over the occlusal surfaces of lower teeth. 

Then upward movement is applied by the thumb, and concomitant inferior force is given by other fngers with pivoting  

the wrist. The advantage of this technique is that it utilizes  

the force created by the muscles of mastication rather than  

overcoming this force as in Nelaton’s technique. 

64.6.1.4 Extraoral Method [8] (Fig. 64.9a, b)

Intraoral methods described previously have the risk of  

human bite in which there are chances of infection transmission. To overcome this, extraoral method has been described.  

In the dislocated mandible, coronoid process comes forward  

which is easy to palpate. On one side, the thumb is positioned over the coronoid process which pushes the mandible  

backward, while the other fngers are located over the mastoid process to deliver counteracting force (Fig. 64.9a). On  

the other side, the mandible is pulled further forward with  

the thumb on the malar eminence and rest of the fngers on  

the mandibular angle (Fig. 64.9b). Pulling the mandible on  

one side with simultaneous pushing of the mandible on the  

other side reduces the dislocation on one side frst and then  

subsequently on the other side. This technique is applicable  

in unilateral dislocation as one side is reduced frst and then  

the other is reduced thereafter













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